Science 302: 611 (2003)

Healthy Animals with Extreme Longevity

Nuno Arantes-Oliveira, Jennifer R. Berman, Cynthia Kenyon

Department of Biochemistry and Biophysics, Mission Bay Genentech Hall, 600 16th Street, Room S312D, University of California, San Francisco, San Francisco, CA 94143Ð2200, USA.

In the nematode Caenorhabditis elegans , mutations that inhibit insulin/IGF-1 (insulin-like growth factor 1) signaling, such as daf-2 insulin/IGF-1 receptor mutations, can double the life-span of the animal. Removing the germ-line precursor cells also extends life-span by approximately 60%. This life-span extension is not a result of sterility; it appears to be due to altered endocrine signaling. Removal of the germ line or the entire reproductive system of daf-2 mutants can further extend life-span: these animals can live four times as long as normal.